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Its main mechanism of action is based on increasing the expression of BDNF (Brain-Derived Neurotrophic Factor) and its TrkB receptors in the hippocampus and prefrontal cortex, promoting synaptic plasticity, long-term potentiation (LTP), and memory consolidation (Dolotov et al., Neuroscience 2006)

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The therapeutic potential of apigenin

Beyond metabolic benefits, recent studies also indicate that IF may moderate immune dysregulation by influencing critical inflammatory signaling pathways, which could have therapeutic implications for autoimmune diseases [59]